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Death is caused by paralysis of respiratory muscles. Injected intramuscularly at minuscule dosage, botulinum toxin type A is used to treat blepharospasm, strabismus, achalasia of the lower esophageal sphincter, and spastic aphonia. A pathological rise in serum Mg2+ levels also causes inhibition of ACh release, hence inhibition of neuromuscular transmission.
Unlike inhalational agents, most of these drugs affect consciousness only and are devoid of analgesic activity (exception: ketamine). The effect cannot be ascribed to nonselective binding to neuronal cell membranes, although this may hold for propofol. Most injectable anesthetics are characterized by a short duration of action. |
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